Disabled protein plays role in progression of some forms of ALS, study finds

March 31, 2015 3:48 PM

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Disabled protein plays role in progression of some forms of ALS, study finds

Neurological conditions like Amyotrhophic lateral sclerosis (ALS) have long been linked to inflammation, but their association hasn’t been clear. Now, researchers at the Icahn School of Medicine at Mount Sinai have found a clue: They discovered that genetic changes may debilitate a protein linked to ALS, rendering it unable to moderate inflammation and thus exacerbate the progression of disease.

The study, published Monday in the journal Nature Immunology, analyzed the relationship of the protein, called senataxin, with the body’s antiviral response, according to a news release.

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